Abstract
Purpose of Review: The outcome of allogeneic stem cell transplantation (allo-HCT) is still compromised by relapse and complications. NK cells and γδT cells, effectors that both function through MHC-unrestricted mechanisms, can target transformed and infected cells without inducing graft-versus-host disease (GVHD). Allo-HCT platforms based on CD34+ selection or αβ-TCR depletion
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