Abstract
Aims: Recent evidence supports the activation of mechanisms underlying cellular ageing and neurodegeneration in developmental lesions associated with epilepsy. The present study examined the ongoing cell injury and vulnerability to neuronal degeneration in glioneuronal tumours (GNT). Methods: We evaluated a series of GNT (n=31 gangliogliomas, GG and n=30 dysembryoplastic neuroepithelial
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