Abstract
Human herpesvirus type 6 (HHV6) is known to reactivate after hematopoetic stem cell transplantation
(HSCT) and has been suggested to be associated with increased mortality and severe clinical manifestations,
including graft versus host disease (GvHD). The exact etiological role of HHV6 reactivation in
increased morbidity and mortality after HSCT remains unclear. This review
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