Abstract
Paraneoplastic cerebellar degeneration (PCD) is one of the most frequent paraneoplastic syndromes affecting the CNS. It is associated with antibodies targeting intracellular neuronal antigens (Hu, Yo, Ri, CV2/CRMP5), which are not thought to be directly pathogenic, and surface antigens (DNER, mGluR1, VGCC), which are potentially pathogenic.1,2 However, in many patients
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